by ElixirThe response: "Driven. Intelligent. Curious. Also, a bad comforter. Lol"
One of these, eh?
No. I haven't grown that distinctive angel wing pattern. It's not thick or too coarse.
And yes, raspberry keytones, somehow suppresses my body hair growth. I mean it will stop growing for days/weeks. Then all of a sudden it starts again. Very strange.
I am fairly sure the hair on my head will never grow past my breast plate. Or if it does, it's taken 7+ years to get there and doesn't seem to want to go any further. Maybe I am impatient.
Um, no, and never. I don't know what they are eating.
Interesting.
http://en.wikipedia.org/wiki/Hypertrichosis
The excess growth of hair occurs in areas of the skin with the exception of androgen-dependent hair of the pubic area, face, and axillary regions.[5]
So body hair is androgen independent or something.
Sounds backwards to me.
raspberry ketone and its derivatives, on antiandrogenic activity
http://www.ncbi.nlm.nih.gov/pubmed/20226658
I don't know about it. But the keytones work, but I'm sure there are long term negative effects or something. I dunno.
What ever those guys are eating, I bet it's an androgen agonist.
https://escholarship.org/uc/item/6gz420mw
Drugs producing increase in hair growth:
Drugs may increase growth of androgen-dependent hairs (hirsutism) or of all hair (hypertrichosis). Hirsutism can be caused by testosterone, danazol, ACTH, metyrapone, anabolic steroids, glucocorticoids and some antiepileptics - phenytoin and carbamazepine.[47] Hypertrichosis can be produced by cyclosporine, minoxidil and diazoxide. Minoxidil and diazoxide open potassium channels in cell membranes leading to hyperpolarisation. The opening of potassium channels could be main mechanism of their hypertrihotic action. Furthermore, it has been shown that other drugs which open potassium channels (P-1075, cromakalim) are able to produce hypertrihosis.[48]